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Receptor Tyrosine Phosphatase

催化受体蛋白去磷酸化的跨膜酶,通过去除酪氨酸磷酸基团调控细胞信号转导,在细胞生长、分化和免疫应答中起关键作用。

产品号 产品名称 蛋白编码 图片
ALP6615

Canine TrkA Protein, Canine, HEK293, N-6His

重组蛋白Canine TrkA Protein, Canine, HEK293, N-6His

A0A8I3P078/ XP_851619.3 (P285-K410) 重组蛋白Canine TrkA Protein, Canine, HEK293, N-6His
TrkA 蛋白 (isoform 3) 属于神经生长因子受体家族,可促进血管生成,过表达时具有致癌活性。TrkA 蛋白 (isoform 3) 可拮抗促进神经元前体细胞分化的抗增殖 NGF-NTRK1 信号。
ALP8080

Human AXL Protein,Human,C-hFc,Expi293

重组蛋白Human AXL Protein,Human,C-hFc,Expi293

P30530 -1 (A26-P449) 重组蛋白Human AXL Protein,Human,C-hFc,Expi293
Axl, a member of the TAM (Tyro3, Axl, Mer) family, and its inhibitors can specifically break the kinase signaling nodes, allowing advanced patients to regain drug sensitivity with improved therapeutic efficacy. Overexpression and activation of Axl receptor tyrosine kinase have been widely accepted to promote cell proliferation, chemotherapy resistance, invasion, and metastasis in several human cancers, such as lung, breast, and pancreatic cancers.
ALP8081

Human AXL Protein,Human,C-His-Avi,Expi293

重组蛋白Human AXL Protein,Human,C-His-Avi,Expi293

P30530 -1 (A26-P449) 重组蛋白Human AXL Protein,Human,C-His-Avi,Expi293
Axl, a member of the TAM (Tyro3, Axl, Mer) family, and its inhibitors can specifically break the kinase signaling nodes, allowing advanced patients to regain drug sensitivity with improved therapeutic efficacy. Overexpression and activation of Axl receptor tyrosine kinase have been widely accepted to promote cell proliferation, chemotherapy resistance, invasion, and metastasis in several human cancers, such as lung, breast, and pancreatic cancers.
ALP8078

Cynomolgus AXL Protein,Cynomolgus,C-His,Expi293

重组蛋白Cynomolgus AXL Protein,Cynomolgus,C-His,Expi293

A0A1D5Q330 (E33-P449) 重组蛋白Cynomolgus AXL Protein,Cynomolgus,C-His,Expi293
Axl, a member of the TAM (Tyro3, Axl, Mer) family, and its inhibitors can specifically break the kinase signaling nodes, allowing advanced patients to regain drug sensitivity with improved therapeutic efficacy. Overexpression and activation of Axl receptor tyrosine kinase have been widely accepted to promote cell proliferation, chemotherapy resistance, invasion, and metastasis in several human cancers, such as lung, breast, and pancreatic cancers.
ALP23732

Recombinant CD45 Protein,Mouse,HEK293,C-Fc

重组蛋白CD45 Protein,Mouse,HEK293,C-Fc

NP_035340.3(Q26-N425) 重组蛋白CD45 Protein,Mouse,HEK293,C-Fc
The CD45 protein is a multifunctional member of the protein tyrosine phosphatase family with heparan sulfate proteoglycan and heparin binding activity as well as protein tyrosine phosphatase activity. It plays a crucial role in lymphocyte differentiation, macromolecular metabolic processes, and signal transduction regulation.
ALP25594

Recombinant IA2 Protein,Human 293a.a,E. coli,N-His

重组蛋白IA2 Protein,Human 293a.a,E. coli,N-His

Q16849-1(M687-Q979) 重组蛋白IA2 Protein,Human 293a.a,E. coli,N-His
The IA2 protein is critical in the vesicle-mediated secretion process and is essential for the accumulation of secretory vesicles in the hippocampus, pituitary gland, and pancreatic islets. IA2 is critical for insulin-related functions, ensuring normal levels of insulin-containing vesicles, preventing degradation and promoting glucose-stimulated insulin secretion.
ALP24345

Recombinant IA2 Protein,Human,E. coli,N-His

重组蛋白IA2 Protein,Human,E. coli,N-His

Q16849-2(R576-Q950) 重组蛋白IA2 Protein,Human,E. coli,N-His
The IA2 protein is critical in the vesicle-mediated secretion process and is essential for the accumulation of secretory vesicles in the hippocampus, pituitary gland, and pancreatic islets. IA2 is critical for insulin-related functions, ensuring normal levels of insulin-containing vesicles, preventing degradation and promoting glucose-stimulated insulin secretion.
ALP23631

Recombinant CD45 Protein,Mouse,HEK293,C-His

重组蛋白CD45 Protein,Mouse,HEK293,C-His

P06800-4(Q26-K566) 重组蛋白CD45 Protein,Mouse,HEK293,C-His
CD45 is a transmembrane glycoprotein , expressed on almost all haematopoietic cells except for mature erythrocytes. PTPRC is an essential regulator of T and B cell antigen receptor-mediated activation.